Changes of NF-&kappa;B, p53, Bcl-2 and caspase in apoptosis induced by JTE-522 in human gastric adenocarcinoma cell line AGS cells: role of reactive oxygen species

doi:10.3748/wjg.v8.i3.431

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Jun 15, 2002 (publication date) through May 9, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

Gastric Cancer

World J Gastroenterol. Jun 15, 2002; 8(3): 431-435
Published online Jun 15, 2002. doi: 10.3748/wjg.v8.i3.431

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Figure 4 Activation of caspase-3 activities by JTE-522 in AGS cells for indicated time period. JTE-522 treatment (0.75 mmol/L) induced cleavage of Ac-DEVD-AMC and Ac-IETD-AMC, indicating activation of caspase-3 activity, respectively

Citation: Li HL, Chen DD, Li XH, Zhang HW, Lü YQ, Ye CL, Ren XD. Changes of NF-κB, p53, Bcl-2 and caspase in apoptosis induced by JTE-522 in human gastric adenocarcinoma cell line AGS cells: role of reactive oxygen species. World J Gastroenterol 2002; 8(3): 431-435
URL: https://www.wjgnet.com/1007-9327/full/v8/i3/431.htm
DOI: https://dx.doi.org/10.3748/wjg.v8.i3.431