Copyright: ©Author(s) 2026.
World J Gastroenterol. Jul 14, 2026; 32(26): 119574
Published online Jul 14, 2026. doi: 10.3748/wjg.v32.i26.119574
Published online Jul 14, 2026. doi: 10.3748/wjg.v32.i26.119574
Figure 3 The relationship between cyclin-dependent kinase 5 expression and the markers Ki-67, programmed death ligand-1, human epidermal growth factor receptor 2, Epstein-Barr virus, and mismatch repair status.
A: Representative images; B: Oncoprint depicting survival status, pathological tumor-node-metastasis stage, and molecular marker profiles across individual cases; C-G: Bar graphs illustrating the distribution of cyclin-dependent kinase 5-low and high groups. PD-L1: Programmed death ligand-1; CPS: Combined positive score; HER2: Human epidermal growth factor receptor 2; EBV: Epstein-Barr virus; MMR: Mismatch repair; pTNM: Pathological tumor-node-metastasis; CDK5: Cyclin-dependent kinase 5.
- Citation: Sun YQ, Su J, Ye KN, Chen QX, Kang EZ, Lv CB, Lian MQ, Zeng WM, Zhang YB, Cai LS. High cyclin-dependent kinase 5 expression promotes tumor progression in gastric neuroendocrine carcinoma. World J Gastroenterol 2026; 32(26): 119574
- URL: https://www.wjgnet.com/1007-9327/full/v32/i26/119574.htm
- DOI: https://dx.doi.org/10.3748/wjg.v32.i26.119574