Copyright: ©Author(s) 2026.
World J Gastroenterol. May 21, 2026; 32(19): 113022
Published online May 21, 2026. doi: 10.3748/wjg.v32.i19.113022
Published online May 21, 2026. doi: 10.3748/wjg.v32.i19.113022
Figure 2 Inducible deletion of Serca2 in the Bmi1+ populations in the gastric tract.
Cryosections prepared from DSerca2Bmi1homo and control (Bmi1-DTmt) mice [0 day-7 days post-tamoxifen (Tx)]; samples were stained for Ki67 analysis and counterstained with 4’,6-diamidino-2-phenylindole. A: Cryosections from esophagus and stomach, were prepared from 5 days post-Tx mice (n = 4); B: Samples from small intestine, 0 day-7 days post-Tx (n = 3) were prepared (several sections were analyzed for each animal). Representative images. Scale bars: 100 μm.
- Citation: Herrero D, Docavo A, Salvador M, Higuera M, Cortés A, García-Ceca J, Montero-Herradón S, García-Brenes MA, Albericio G, Cordero A, Carmona RM, de Molina A, Salvador JM, Mora C, Zapata AG, Bernad A. Serca2 deletion in the mouse adult Bmi1+ compartment induces a lethal phenotype involving a severe gastric dysfunction. World J Gastroenterol 2026; 32(19): 113022
- URL: https://www.wjgnet.com/1007-9327/full/v32/i19/113022.htm
- DOI: https://dx.doi.org/10.3748/wjg.v32.i19.113022