Helicobacter pylori eradication and the prevention of peptic ulcer bleeding and cardiovascular disease progression in chronic aspirin users

Table 2 Endoscopic and histopathological findings in study participants, n (%)

Category	Variable	H. pylori-positive (n = 262)	H. pylori-negative (n = 138)	P value
Endoscopic	Normal	122 (46.6)	136 (98.6)	< 0.001
	Atrophic gastritis	62 (23.7)	0 (0.0)	< 0.001
	Gastric erosion	34 (12.2)	0 (0.0)	< 0.001
	Gastric ulcer	28 (10.7)	0 (0.0)	< 0.001
	Antral erosion	6 (2.3)	0 (0.0)	0.378
	Duodenal ulcer	6 (2.3)	0 (0.0)	0.378
	Duodenitis	6 (2.3)	0 (0.0)	0.378
Histopathological	H. pylori colonization	262 (100)	0 (0.0)	< 0.001
	Inflammation severity
	Mild	80 (30.5)	40 (29.0)	0.75
	Moderate	120 (45.8)	30 (21.7)	< 0.001
	Severe	50 (19.1)	0 (0.0)	< 0.001
	Atrophy	40 (15.3)	0 (0.0)	< 0.001
	Intestinal metaplasia	20 (7.6)	0 (0.0)	0.002

Gastric ulcers and duodenal ulcers are subtypes of peptic ulcers, differentiated by their location in the stomach and duodenum, respectively. A P value < 0.05 was considered statistically significant. H. pylori: Helicobacter pylori.