Mitochondrial dysfunction as a bridge to pathology in acute pancreatitis: From molecular insights to novel therapeutic strategies

doi:10.3748/wjg.v31.i48.113840

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Dec 28, 2025; 31(48): 113840
Published online Dec 28, 2025. doi: 10.3748/wjg.v31.i48.113840

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Figure 1 Mitochondrial structure and function in pancreatic acinar cells. Cyp D: Cyclophilin D; ANT: Adenine nucleotide translocation; mtDNA: Mitochondrial deoxyribonucleic acid; SERCA: Sarco/endoplasmic reticulum Ca²⁺-ATPases; PMCA: Plasma membrane Ca²⁺-ATPases.

Citation: Xia CC, Xu Y, Wang ZH, Xu GQ. Mitochondrial dysfunction as a bridge to pathology in acute pancreatitis: From molecular insights to novel therapeutic strategies. World J Gastroenterol 2025; 31(48): 113840
URL: https://www.wjgnet.com/1007-9327/full/v31/i48/113840.htm
DOI: https://dx.doi.org/10.3748/wjg.v31.i48.113840

Xia CC, Xu Y, Wang ZH, Xu GQ. Mitochondrial dysfunction as a bridge to pathology in acute pancreatitis: From molecular insights to novel therapeutic strategies. World J Gastroenterol 2025; 31(48): 113840 [PMID: 41480323 DOI: 10.3748/wjg.v31.i48.113840]

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