Role of cell-free network communication in alcohol-associated disorders and liver metastasis

doi:10.3748/wjg.v27.i41.7080

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World Journal of Gastroenterology

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1007-9327

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World J Gastroenterol. Nov 7, 2021; 27(41): 7080-7099
Published online Nov 7, 2021. doi: 10.3748/wjg.v27.i41.7080

Table 2 Extracellular vesicle components in cancer progression and metastasis

EV cargo	Type of molecule	Action on recipient cells/tissue	Type of cancer	Ref.
CEA	Protein	Inflammation	Colorectal	Yokoyama et al[162]
KRAS	Protein	Invasiveness in recipient cells	Colorectal	Beckler et al[152]
ITG	Protein	Metastatic organotropism	Breast	Hoshino et al[96]
TNC	ECM protein	Stem cell niche formation	Breast	Oskarsson et al[97]
MIF	Protein	Liver premetastatic niche formation	Pancreatic	Costa-Silva et al[168]
ZFAS1	lncRNA	Cancer growth/metastasis	Gastric	Pan et al[119]
Amino acids, lipids, TCA-cycle intermediates	Metabolites	Cancer growth	Prostate	Zhao et al[132]

CEA: Carcinoembryonic antigen; KRAS: Kirsten rat sarcoma viral oncogene; ITG: Integrins; TNC: Tenascin C; MIF: Macrophage migration inhibitory factor; ZFAS1: ZNFX1 antisense RNA1; LncRNA: Long non-coding RNA; ECM: Extracellular matrix.

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Citation: Kuracha MR, Thomas P, Tobi M, McVicker BL. Role of cell-free network communication in alcohol-associated disorders and liver metastasis. World J Gastroenterol 2021; 27(41): 7080-7099
URL: https://www.wjgnet.com/1007-9327/full/v27/i41/7080.htm
DOI: https://dx.doi.org/10.3748/wjg.v27.i41.7080

Kuracha MR, Thomas P, Tobi M, McVicker BL. Role of cell-free network communication in alcohol-associated disorders and liver metastasis. World J Gastroenterol 2021; 27(41): 7080-7099 [PMID: 34887629 DOI: 10.3748/wjg.v27.i41.7080]

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