Herbs-partitioned moxibustion alleviates aberrant intestinal epithelial cell apoptosis by upregulating A20 expression in a mouse model of Crohn’s disease

Figure 4 Expression levels of A20, tumor necrosis factor alpha, tumor necrosis factor receptor 1, tumor necrosis factor receptor 1-associated death domain, receptor-interacting protein 1, and FAS-associated death domain protein across groups. Data are presented as the mean ± standard deviation (n = 10). Data were evaluated for statistical significance using one-way analysis of variance and are represented as follows: ^aP < 0.05, ^bP < 0.01 as compared to normal contro; ^cP < 0.05, ^dP < 0.01 as compared to model control; ^eP < 0.05, ^fP < 0.01 as compared to mesalazine; ^gP < 0.05, ^hP < 0.01 as compared to wild type. TNFR1: Tumor necrosis factor receptor 1; RIP1: Receptor-interacting protein 1; TNF-α: Tumor necrosis factor alpha; TRADD: Tumor necrosis factor receptor 1-associated death domain; FADD: FAS-associated death domain; WT: Wild type; NC: Normal control; MC: Model control; MESA: Mesalazine; HPM: Herbs-partitioned moxibustion.