MicroRNA-155 promotes the pathogenesis of experimental colitis by repressing SHIP-1 expression

doi:10.3748/wjg.v23.i6.976

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Feb 14, 2017 (publication date) through Apr 17, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Feb 14, 2017; 23(6): 976-985
Published online Feb 14, 2017. doi: 10.3748/wjg.v23.i6.976

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Figure 4 Effects of antagomiR-155 on dextran sulfate sodium-induced histological changes in intestinal mucosa. A: Representative histological images of colon tissues from mice in differently treated groups (original magnification, upper panel, × 200; lower panel, × 400); B: Microscopic inflammatory infiltration score. ^bP < 0.01, DSS vs control; ^aP < 0.05, DSS + antagomiR-155 vs DSS + NC; C: Microscopic crypt destruction score. ^bP < 0.01, DSS vs control. DSS: Dextran sulfate sodium.

Citation: Lu ZJ, Wu JJ, Jiang WL, Xiao JH, Tao KZ, Ma L, Zheng P, Wan R, Wang XP. MicroRNA-155 promotes the pathogenesis of experimental colitis by repressing SHIP-1 expression. World J Gastroenterol 2017; 23(6): 976-985
URL: https://www.wjgnet.com/1007-9327/full/v23/i6/976.htm
DOI: https://dx.doi.org/10.3748/wjg.v23.i6.976