MicroRNA-155 promotes the pathogenesis of experimental colitis by repressing SHIP-1 expression

doi:10.3748/wjg.v23.i6.976

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Feb 14, 2017 (publication date) through Apr 24, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Feb 14, 2017; 23(6): 976-985
Published online Feb 14, 2017. doi: 10.3748/wjg.v23.i6.976

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Figure 3 Inhibition of microRNA-155 alleviated mouse experimental colitis. A: AntagomiR-155 treatment significantly reduced the expression level of miR-155 in colon tissues. ^bP < 0.01, dextran sulfate sodium (DSS) vs control, DSS + antagomiR-155 vs DSS + NC; B: Inhibition of miR-155 markedly decreased the disease activity index of experimental colitis. ^aP < 0.05, DSS + antagomiR-155 vs DSS + NC, DSS; C: AntagomiR-155 treatment alleviated the shortening of colon induced by DSS.

Citation: Lu ZJ, Wu JJ, Jiang WL, Xiao JH, Tao KZ, Ma L, Zheng P, Wan R, Wang XP. MicroRNA-155 promotes the pathogenesis of experimental colitis by repressing SHIP-1 expression. World J Gastroenterol 2017; 23(6): 976-985
URL: https://www.wjgnet.com/1007-9327/full/v23/i6/976.htm
DOI: https://dx.doi.org/10.3748/wjg.v23.i6.976