Reprogramming of glucose metabolism in hepatocellular carcinoma: Progress and prospects

doi:10.3748/wjg.v22.i45.9933

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Dec 7, 2016; 22(45): 9933-9943
Published online Dec 7, 2016. doi: 10.3748/wjg.v22.i45.9933

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Figure 1 Reprogramming of glucose metabolism in hepatocellular carcinoma. Reprogramming of glucose metabolism-related enzymes and transporting proteins in HCC. The expression of GLUT1, GLUT2, HK2, HKDC1, GAPDH, PKM2, LDHA and MCT4 are up-regulated in HCC glycolysis pathway. GLUT: Glucose transporter; HK: Hexokinase; G6P: Glucose-6-phosphate; GPI1: Glucose-6-phosphate isomerase 1; F6P: Fructose-6-phosphate; PFK: Phosphofructokinase; FBP: Fructose-1,6-bisphosphatase; ALDA: Aldolase A, DHAP: Dihydroxyacetone phosphate; TIM: Triosephosphate isomerase; G3P: Glyceraldehyde-3-phosphate; GAPDH: Glyceraldehyde-3-phosphate dehydrogenase; PG: Phosphoglycerate; PGAM: Phosphoglycerate mutase; ENO: Enolase; PEP: Phosphoenolpyruvate; PKM2: Pyruvate kinase isoform M2; PFK: Phosphate fructose kinase; LDHA: Lactate dehydrogenase A; MCT4: Monocarboxylate transporter 4.

Citation: Shang RZ, Qu SB, Wang DS. Reprogramming of glucose metabolism in hepatocellular carcinoma: Progress and prospects. World J Gastroenterol 2016; 22(45): 9933-9943
URL: https://www.wjgnet.com/1007-9327/full/v22/i45/9933.htm
DOI: https://dx.doi.org/10.3748/wjg.v22.i45.9933