Hepatitis C virus NS5A promotes insulin resistance through IRS-1 serine phosphorylation and increased gluconeogenesis

doi:10.3748/wjg.v21.i43.12361

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Nov 21, 2015 (publication date) through Apr 18, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Nov 21, 2015; 21(43): 12361-12369
Published online Nov 21, 2015. doi: 10.3748/wjg.v21.i43.12361

Table 1 Primers used in the study

Gene	Forward primer	Reverse primer
CREB	GATCTTAGTGCCCAGCAACC	GACGGACCTCTCTCTTTCGT
PEPCK	GGCTACAACTTCGGCAAATACC	GGAAGATCTTGGGCAGTTTGG
G6P	CATTGACACCACACCCTTTGC	CCCTGTACATGCTGGAGTTGAG
TNF-α	AGGCGCTCCCCAAGAAGACA	TCCTTGGCAAAACTGCACCT

TNF-α: Tumor necrosis factor-alpha; PEPCK: Phosphoenol pyruvate carboxykinase; G6P: Glucose-6-phosphatase; CREB: CRE-binding protein.

Citation: Parvaiz F, Manzoor S, Iqbal J, Sarkar-Dutta M, Imran M, Waris G. Hepatitis C virus NS5A promotes insulin resistance through IRS-1 serine phosphorylation and increased gluconeogenesis. World J Gastroenterol 2015; 21(43): 12361-12369
URL: https://www.wjgnet.com/1007-9327/full/v21/i43/12361.htm
DOI: https://dx.doi.org/10.3748/wjg.v21.i43.12361