Arterial structure and function in inflammatory bowel disease

Table 1 Studies that have measured carotid-femoral pulse wave velocity in subjects with inflammatory bowel disease

Ref.	Anti TNF-αtherapy (%)	Subjects, n			Pulse wave velocity (m/s)
		IBD		Controls	IBD		Controls
		CD	UC		CD	UC
Zanoli et al[25] (2012)	13	16	16	32	6.5 ± 1.5	6.8 ± 1.3	6.0 ± 0.8a
Akdogan et al[26] (2013)	5	0	37	30	-	8.9 ± 3.0	7.2 ± 1.7c
Theocharidou et al[31] (2013)	44	43	23	44	6.8 ± 1.3	6.3 ± 1.1	6.1 ± 0.9
Zanoli et al[29] (2014)	19	34	40	80	8.0 ± 1.6	7.8 ± 1.7	7.0 ± 1.1bcd
Korkmaz et al[27] (2014)	2	18	84	74	6.4 ± 1.2	6.6 ± 1.2	5.9 ± 1.2a
Theocharidou et al[31] (2014)	46	29	15	44	7 ± 1.2	6.3 ± 1.2d	6.4 ± 0.9d
Aytaç et al[28] (2015)	0	25	30	25	9.6 ± 1.4	9.3 ± 1.3	7.6 ± 0.3ef

Data are presented as percentages (%), counts or mean ± SD.

^aP < 0.05 vs the whole group of subjects with IBD;

^bP < 0.001 vs the whole group of subjects with IBD;

^cP < 0.05 vs subjects with ulcerative colitis;

^dP < 0.001 vs subjects with Crohn’s disease;

^eP < 0.05 vs subjects with ulcerative colitis;

^fP < 0.001 vs subjects with Crohn’s disease. CD: Crohn’s disease; UC: Ulcerative colitis; IBD: Inflammatory bowel disease.