Role of abnormal lipid metabolism in development, progression, diagnosis and therapy of pancreatic cancer

doi:10.3748/wjg.v20.i9.2279

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Mar 7, 2014 (publication date) through Apr 24, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Mar 7, 2014; 20(9): 2279-2303
Published online Mar 7, 2014. doi: 10.3748/wjg.v20.i9.2279

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Figure 6 Possible role of aldo-keto reductase 1B10 in regulation of cell proliferation in pancreatic cancer. Binding of AKR1B10 results in stabilization of ACCA, and up-regulation of fatty acid synthesis. AKR1B10: Aldo-keto reductase family 1B10; ACCA: Acetyl-CoA carboxylase; Isopentenyl-PP: Isopentenyl diphosphate; Geranyl-PP: Geranyl diphosphate; Farnesyl-PP: Farnesyl diphosphate; Geranylgeranyl-PP: Geranylgeranyl diphosphate.

Citation: Swierczynski J, Hebanowska A, Sledzinski T. Role of abnormal lipid metabolism in development, progression, diagnosis and therapy of pancreatic cancer. World J Gastroenterol 2014; 20(9): 2279-2303
URL: https://www.wjgnet.com/1007-9327/full/v20/i9/2279.htm
DOI: https://dx.doi.org/10.3748/wjg.v20.i9.2279