Interactions between pork consumption, CagA status and IL-1B-31 genotypes in gastric cancer

doi:10.3748/wjg.v20.i25.8151

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Jul 7, 2014; 20(25): 8151-8157
Published online Jul 7, 2014. doi: 10.3748/wjg.v20.i25.8151

Table 1 Demographic data of cases and controls n (%)

Characteristic		Cases	Controls	t/χ²	P value
Characteristic		n = 171	n = 367
Age (yr, mean ± SD)		56.93 ± 14.01	56.81 ± 13.90	0.093¹	0.440
Gender
	Male	118 (69.01)	243 (66.21)	0.412	0.521
	Female	53 (30.99)	124 (33.79)	0.412	0.521
Education
	Primary	51 (29.82)	117 (31.88)	0.332	0.847
	Secondary	84 (49.12)	179 (48.77)
	Tertiary and postgraduate	36 (21.06)	71 (19.35)
BMI (kg/m²)
	≤ 25	110 (64.33)	244 (66.49)	0.365	0.794
	> 25	61 (35.67)	123 (33.51)	0.365	0.794
H. pylori CagA positive		101 (59.06)	163 (44.41)	10.018	0.002^a
IL-1B-31
	TT	41 (23.98)	89 (24.25)	0.005	0.997
	TC	84 (49.12)	180 (49.05)
	CC	46 (26.90)	98 (26.70)
	C carrier	130 (76.02)	278 (75.75)
Pork consumption
	< 25 g/d	89 (52.05)	177 (48.23)	0.680	0.410
	≥ 25 g/d	82 (47.95)	190 (51.77)	0.680	0.410

¹For t test.

^aP < 0.05 vs control group. BMI: Body mass index; H. pylori: Helicobacter pylori; IL: Interleukin.

Citation: Wang XQ, Terry PD, Cheng L, Yan H, Wang JS, Wu WA, Hu SK. Interactions between pork consumption, CagA status and IL-1B-31 genotypes in gastric cancer. World J Gastroenterol 2014; 20(25): 8151-8157
URL: https://www.wjgnet.com/1007-9327/full/v20/i25/8151.htm
DOI: https://dx.doi.org/10.3748/wjg.v20.i25.8151