Pathogenesis of NSAID-induced gastric damage: Importance of cyclooxygenase inhibition and gastric hypermotility

doi:10.3748/wjg.v18.i18.2147

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May 14, 2012 (publication date) through Apr 24, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. May 14, 2012; 18(18): 2147-2160
Published online May 14, 2012. doi: 10.3748/wjg.v18.i18.2147

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Figure 3 Representative recordings showing the effects of various non-steroidal anti-inflammatory drug on gastric motility in rats. Indomethacin (35 g/kg), aspirin (200 mg/kg), naproxen (40 mg/kg), flurbiprofen (20 mg/kg) or diclofenac (40 mg/kg) was given s.c. after basal motility had stabilized (data from ref.18 after modification).

Citation: Takeuchi K. Pathogenesis of NSAID-induced gastric damage: Importance of cyclooxygenase inhibition and gastric hypermotility. World J Gastroenterol 2012; 18(18): 2147-2160
URL: https://www.wjgnet.com/1007-9327/full/v18/i18/2147.htm
DOI: https://dx.doi.org/10.3748/wjg.v18.i18.2147