A potential oncogenic role of the commonly observed E2F5 overexpression in hepatocellular carcinoma

doi:10.3748/wjg.v17.i4.470

Advanced Search

BPG is committed to discovery and dissemination of knowledge

Home / Archive / Volume 17, Issue 4

This Article

Citation of this article

Corresponding Author of This Article

Article-Type of This Article

Open-Access Policy of This Article

Times Cited Counts in Google of This Article

Number of Hits and Downloads for This Article

Total Article Views (6328)

All Articles published online

The chart showing PDF series, HTML series, Figures (1-6) series.

Item

Count

PDF

491

HTML

5676

Figures (1-6)

161

Sum=6328

Jan 28, 2011 (publication date) through Apr 24, 2024

Times Cited of This Article

Times Cited (38)

Journal Information of This Article

Publication Name

World Journal of Gastroenterology

ISSN

1007-9327

Publisher of This Article

Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

Original Article

World J Gastroenterol. Jan 28, 2011; 17(4): 470-477
Published online Jan 28, 2011. doi: 10.3748/wjg.v17.i4.470

Full Size Figure Download Figure

Figure 4 Effects of E2F5 knockdown on HepG2 cell growth. Proliferation assay (A), colony formation assay (B), and soft agar assay (C) consistently demonstrated the inhibition of HepG2 cell growth by E2F5-specific small interfering RNA transfection (× 40).

Citation: Jiang Y, Yim SH, Xu HD, Jung SH, Yang SY, Hu HJ, Jung CK, Chung YJ. A potential oncogenic role of the commonly observed E2F5 overexpression in hepatocellular carcinoma. World J Gastroenterol 2011; 17(4): 470-477
URL: https://www.wjgnet.com/1007-9327/full/v17/i4/470.htm
DOI: https://dx.doi.org/10.3748/wjg.v17.i4.470