Gastroesophageal flap valve status distinguishes clinical phenotypes of large hiatal hernia

doi:10.3748/wjg.v16.i47.6010

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Dec 21, 2010 (publication date) through Apr 18, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Dec 21, 2010; 16(47): 6010-6015
Published online Dec 21, 2010. doi: 10.3748/wjg.v16.i47.6010

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Figure 3 Representative endoscopic images of five patients with Cameron lesions. A: Case #1 (no nonsteroidal anti-inflammatory drug, Hb5.4); B: Case #2 (loxoprofen + low dose aspirin, Hb5.3); C: Case #3 (diclofenac, Hb6.3); D, E: Case #4 (low dose aspirin, Hb8.0); F, G: Case #5 (low dose aspirin, Hb10.3). An ulceration scar was noted at the point at which the gastroesophageal flap valve fold diverged from the anterior stomach wall (arrows in E and F).

Citation: Kaneyama H, Kaise M, Arakawa H, Arai Y, Kanazawa K, Tajiri H. Gastroesophageal flap valve status distinguishes clinical phenotypes of large hiatal hernia. World J Gastroenterol 2010; 16(47): 6010-6015
URL: https://www.wjgnet.com/1007-9327/full/v16/i47/6010.htm
DOI: https://dx.doi.org/10.3748/wjg.v16.i47.6010