Bile acids as endogenous etiologic agents in gastrointestinal cancer

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Jul 21, 2009 (publication date) through Apr 25, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Jul 21, 2009; 15(27): 3329-3340
Published online Jul 21, 2009. doi: 10.3748/wjg.15.3329

Table 3 Bile acids induce ROS/RNS in colon cells

Cells/tissues	Bile acid(s) that induced ROS/RNS	Ref.
Human colon surgical resections	DCA (RNS)	[110]
Cultured human adenocarcinoma cells (CACO-2)	DCA, LCA (ROS)	[111]
Cultured human adenocarcinoma cells (HT-29)	DCA, LCA (ROS)	[112]
	DCA (ROS)	[36 113]
	DCA (RNS)	[114]
Cultured human adenocarcinoma cells (HCT116)	DCA (ROS)	[109 115 116]
Cultured human adenocarcinoma cells (HCT116)	DCA (RNS)	[117]
Rat colonic mucosa	DCA (ROS)	[118]
Mouse colonic mucosa	DCA (ROS, RNS)	[119]

Citation: Bernstein H, Bernstein C, Payne CM, Dvorak K. Bile acids as endogenous etiologic agents in gastrointestinal cancer. World J Gastroenterol 2009; 15(27): 3329-3340
URL: https://www.wjgnet.com/1007-9327/full/v15/i27/3329.htm
DOI: https://dx.doi.org/10.3748/wjg.15.3329