Bile acids as endogenous etiologic agents in gastrointestinal cancer

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Jul 21, 2009 (publication date) through May 3, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Jul 21, 2009; 15(27): 3329-3340
Published online Jul 21, 2009. doi: 10.3748/wjg.15.3329

Table 2 Bile acids induce apoptosis in liver cells

Cells/tissues	Bile acid(s) that induced apoptosis	Ref.
Isolated rat hepatocytes	GDCA	[64 65]
	GCDCA	[55 66]
	GCDCA, GCA	[57]
	GCDCA	[67]
Isolated rat and mouse hepatocytes	DCA	[68]
Liver tissue sections from rats fed DCA, and cultured human hepatocellular carcinoma cells (HuH-7)	DCA	[58]
Cultured rat hepatocyes (McNtcp.24 cells)	GCDC	[69 70]
Cultured human hepatocellular carcinoma cells (HuH-7)	GCDCA	[71]
Rat hepatocytes and human hepatoma carcinoma cells (HuH-7)	Taurolithocholate-3-sulfate	[59]

Citation: Bernstein H, Bernstein C, Payne CM, Dvorak K. Bile acids as endogenous etiologic agents in gastrointestinal cancer. World J Gastroenterol 2009; 15(27): 3329-3340
URL: https://www.wjgnet.com/1007-9327/full/v15/i27/3329.htm
DOI: https://dx.doi.org/10.3748/wjg.15.3329