©2009 The WJG Press and Baishideng.
World J Gastroenterol. Jun 7, 2009; 15(21): 2570-2578
Published online Jun 7, 2009. doi: 10.3748/wjg.15.2570
Published online Jun 7, 2009. doi: 10.3748/wjg.15.2570
Table 1 Pathophysiology of malnutrition
| Main mechanism | Effect |
| Decreased food intake | Anorexia |
| Abdominal pain, nausea, vomiting | |
| Restricted diets | |
| Drugs | |
| Nutrients malabsorption | Reduced absorptive surface due to inflammation, resection, bypass and fistulae |
| Increased intestinal loss | Exudative enteropathy (protein loss) |
| Occult/overt blood loss (iron deficiency) | |
| Diarrhea (increased loss of Zn2+, K+, Mg2+) | |
| Steatorrhea (fat and fat soluble vitamin malabsorption, and divalent cations’ loss: Zn2+, Mg2+, Ca2+, Cu2+) | |
| Hypermetabolic state | Alterations of resting energy expenditure |
| Drugs' interaction | Anorexia, nausea, test alteration, proteolysis, interaction with nutrients absorption/utilization |
- Citation: Hartman C, Eliakim R, Shamir R. Nutritional status and nutritional therapy in inflammatory bowel diseases. World J Gastroenterol 2009; 15(21): 2570-2578
- URL: https://www.wjgnet.com/1007-9327/full/v15/i21/2570.htm
- DOI: https://dx.doi.org/10.3748/wjg.15.2570