CagA+ H pylori infection is associated with polarization of T helper cell immune responses in gastric carcinogenesis

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Jun 7, 2007 (publication date) through May 3, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Jun 7, 2007; 13(21): 2923-2931
Published online Jun 7, 2007. doi: 10.3748/wjg.v13.i21.2923

Table 1 Sequences of cDNA primers for human IFN-γ, IL-4 and β-actin

Target	Sequences (5’→3’)	Length of amplicon (bp)
IFN-γ	Forward: CTTTAAAGATGACCAGAGCATCCAA	189 (372-560 NM000619.2)
IFN-γ	Reverse: GGCGACAGTTCAGCCATCAC
IL-4	Forward: GACTGTGCTCCGGCAGTTCTA	182 (589-770 NM000589)
IL-4	Reverse: CCAACGTACTCTGGTTGGCTTC
β-actin	Forward: ATTGCCGACAGGATGCAGA	89 (991-1079 CR609136.1)
β-actin	Reverse: GAGTACTTGCGCTCAGGAGGA

Citation: Wang SK, Zhu HF, He BS, Zhang ZY, Chen ZT, Wang ZZ, Wu GL. CagA+ H pylori infection is associated with polarization of T helper cell immune responses in gastric carcinogenesis. World J Gastroenterol 2007; 13(21): 2923-2931
URL: https://www.wjgnet.com/1007-9327/full/v13/i21/2923.htm
DOI: https://dx.doi.org/10.3748/wjg.v13.i21.2923