Pathophysiology of pulmonary complications of acute pancreatitis

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Nov 28, 2006 (publication date) through Apr 23, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Nov 28, 2006; 12(44): 7087-7096
Published online Nov 28, 2006. doi: 10.3748/wjg.v12.i44.7087

Table 3 Pro-inflammatory cytokines

IL-1β -major inflammatory mediator, major activator of macrophages and enhances B and T cell activation

IL-6 -acute phase reactant, stimulates B cell differentiation

IL-8 -stimulates the upregulation of adhesion molecules, chemotactic factor for neutrophils and lymphocytes

TNF-α -recruitment of inflammatory cells, regulation of cytokine production, and promotion of pancreatic acinar cell death by apoptosis

IL-10 is an anti-inflammatory cytokine by inhibiting macrophages releasing inflammator mediators.

Citation: Browne GW, Pitchumoni C. Pathophysiology of pulmonary complications of acute pancreatitis. World J Gastroenterol 2006; 12(44): 7087-7096
URL: https://www.wjgnet.com/1007-9327/full/v12/i44/7087.htm
DOI: https://dx.doi.org/10.3748/wjg.v12.i44.7087