S-adenosyl-methionine decreases ethanol-induced apoptosis in primary hepatocyte cultures by a c-Jun N-terminal kinase activity-independent mechanism

doi:10.3748/wjg.v12.i12.1895

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Mar 28, 2006 (publication date) through May 26, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Mar 28, 2006; 12(12): 1895-1904
Published online Mar 28, 2006. doi: 10.3748/wjg.v12.i12.1895

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Figure 5 Prevention of GSH decrease in hepatocyte cultures exposed to ethanol by AdoMet. Data represent percentage change of ROS levels in mean±SEM of n = 3. ^arepresents a significant difference (P < 0.05) in ROS generation between pretreated cells and cells exposed to ethanol only.

Citation: Cabrales-Romero MDP, Márquez-Rosado L, Fattel-Fazenda S, Trejo-Solís C, Arce-Popoca E, Alemán-Lazarini L, Villa-Treviño S. S-adenosyl-methionine decreases ethanol-induced apoptosis in primary hepatocyte cultures by a c-Jun N-terminal kinase activity-independent mechanism. World J Gastroenterol 2006; 12(12): 1895-1904
URL: https://www.wjgnet.com/1007-9327/full/v12/i12/1895.htm
DOI: https://dx.doi.org/10.3748/wjg.v12.i12.1895