Impaired gallbladder motility and delayed orocecal transit contribute to pigment gallstone and biliary sludge formation in β -thalassemia major adults

Figure 6 Varations in individual fasting gallbladder volumes for each subgroup in β -thalassemia major patients. A: Major changes in patients who developed biliary sludge: asterisks indicate significant differences, compared to the other two groups of patients (^aP < 0.05 vs prior sludge/gallstones group and ^cP < 0.017 vs still anechoic group). B: Percent increased fasting gallbladder volume in the three groups of patients. The last bar represents those gallbladders with stones that developed additional sludge (n = 2).