Adenosine deaminase in pleural effusion: Bridging diagnosis and the pathophysiology of inflammation

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Aug 6, 2025 (publication date) through Jun 26, 2025

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World Journal of Clinical Cases

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2307-8960

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Clin Cases. Aug 6, 2025; 13(22): 106925
Published online Aug 6, 2025. doi: 10.12998/wjcc.v13.i22.106925

Table 4 Key questions for future research

Research question	Potential impact
How does P-ADA correlate with disease severity?	Improves prognostic stratification
Can P-ADA differentiate between specific etiologies (e.g., TB vs cancer)?	Enhances more targeted and effective treatment plans
What is the role of ADA in resolving inflammation?	Identifies new therapeutic targets for inflammatory conditions
How does P-ADA interact with other biomarkers (e.g., IL-6, ADA1)?	Strengthens multimodal diagnostics
Are there genetic variations affecting P-ADA levels?	Explains population-specific differences in cutoffs

TB: Tuberculosis; P-ADA: Pleural adenosine deaminase; IL-6: Interleukin 6; ADA1: Adenosine deaminase 1.

Citation: Shi DD, Tian J, Ding J. Adenosine deaminase in pleural effusion: Bridging diagnosis and the pathophysiology of inflammation. World J Clin Cases 2025; 13(22): 106925
URL: https://www.wjgnet.com/2307-8960/full/v13/i22/106925.htm
DOI: https://dx.doi.org/10.12998/wjcc.v13.i22.106925