African origins and chronic kidney disease susceptibility in the human immunodeficiency virus era

doi:10.5527/wjn.v4.i2.295

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May 6, 2015 (publication date) through Apr 25, 2024

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World Journal of Nephrology

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2220-6124

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Nephrol. May 6, 2015; 4(2): 295-306
Published online May 6, 2015. doi: 10.5527/wjn.v4.i2.295

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Figure 3 Gene-Gene, Gene-Environment steering contribution to APOL1 associated CKD and the positive selection of APOL1 associated CKD variants as a result of Trypanosomiasis. SRA: Serum resistant associated protein; HIV: Human immunodeficiency virus; T.b: Trypanosoma brucei; APOL1: Apolipoprotein L1; MYH9: Non-muscle myosin heavy chain 9; HIVAN: Human immunodeficiency virus-associated nephropathy; FSGS: Focal segmental glomerulosclerosis; T2DM: Type 2 diabetes mellitus; ESRD: End stage renal disease; CKD: Chronic kidney disease.

Citation: Kasembeli AN, Duarte R, Ramsay M, Naicker S. African origins and chronic kidney disease susceptibility in the human immunodeficiency virus era. World J Nephrol 2015; 4(2): 295-306
URL: https://www.wjgnet.com/2220-6124/full/v4/i2/295.htm
DOI: https://dx.doi.org/10.5527/wjn.v4.i2.295