Treatment of hypogonadotropic male hypogonadism: Case-based scenarios

doi:10.5527/wjn.v4.i2.245

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May 6, 2015 (publication date) through Apr 24, 2024

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World Journal of Nephrology

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2220-6124

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Nephrol. May 6, 2015; 4(2): 245-253
Published online May 6, 2015. doi: 10.5527/wjn.v4.i2.245

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Figure 1 Hypogonadism therapeutic hormones and mechanism of action. Exogenous T and anabolic steroids negatively affect the HPG axis. SERMs such as CC stop the negative feedback of estrogen on the HPG axis. hCG stimulates Leydig cells. Aromatase inhibitors inhibit the conversion of T to E. LH: Luteinizing hormone; FSH: Follicle-stimulating hormone; AAS: Anabolic-Androgenic steroid; hCG: Human chorionic gonadotropin; GnRH: Gonadotropin-releasing hormone.

Citation: Crosnoe-Shipley LE, Elkelany OO, Rahnema CD, Kim ED. Treatment of hypogonadotropic male hypogonadism: Case-based scenarios. World J Nephrol 2015; 4(2): 245-253
URL: https://www.wjgnet.com/2220-6124/full/v4/i2/245.htm
DOI: https://dx.doi.org/10.5527/wjn.v4.i2.245