Pathogenesis of glomerular haematuria

doi:10.5527/wjn.v4.i2.185

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May 6, 2015 (publication date) through Sep 17, 2024

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World Journal of Nephrology

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2220-6124

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Nephrol. May 6, 2015; 4(2): 185-195
Published online May 6, 2015. doi: 10.5527/wjn.v4.i2.185

Table 1 Classification of haematuric diseases by histopathological findings

Glomerular endothelial cell layer	GBM disorders	Mesangial deposits	Podocytary slit diaphragm disorders	Subendothelial/subepithelial deposit	Others
ANCA	Primary	IgAN	MYH9 disease	Primary GN	WRN
Endocapillary	Alport	HSP	Fabry disease	MBP	SCD
	TBMD			Endocapillary
	HANAC			Crescentic
	? LPHS
				Secondary GN
	Secondary			SLE
	Anti-GBM disease			Cryoglobulinemia
	C3 glomerulopathy
	CFHR5 nephropathy			Fibrillar deposit
				Fibronectin
				Fibrillary
				Immunotactoid

GBM: Glomerular basement membrane; ANCA: Antineutrophil cytoplasmic antibodies; CFHR5: Complement factor H–related 5 nephropathy; GN: Glomerulonephritis; HANAC: Hereditary angiopathy, nephropathy, aneurysms, and muscle cramps syndrome; IgAN: IgA nephropathy; LPHS: Loin back pain syndrome; MBP: Membranoproliferative; SCD: Sickle cell disease; HSP: Henoch-Schönlein purpura; SLE: Systemic lupus erythematous; TBMD: Thin basement membrane disease; WRN: Warfarin related nephropathy.

Citation: Yuste C, Gutierrez E, Sevillano AM, Rubio-Navarro A, Amaro-Villalobos JM, Ortiz A, Egido J, Praga M, Moreno JA. Pathogenesis of glomerular haematuria. World J Nephrol 2015; 4(2): 185-195
URL: https://www.wjgnet.com/2220-6124/full/v4/i2/185.htm
DOI: https://dx.doi.org/10.5527/wjn.v4.i2.185