Lactylation in pancreatic cancer: From molecular mechanisms to therapeutic perspectives

doi:10.5306/wjco.v17.i3.115882

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World Journal of Clinical Oncology

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2218-4333

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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Copyright: ©Author(s) 2026. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution-NonCommercial (CC BY-NC 4.0) license. No commercial re-use. See permissions. Published by Baishideng Publishing Group Inc.

World J Clin Oncol. Mar 24, 2026; 17(3): 115882
Published online Mar 24, 2026. doi: 10.5306/wjco.v17.i3.115882

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Figure 3 Therapeutic targeting strategies for lactylation in pancreatic ductal adenocarcinoma. ACSS2: Acyl-CoA synthetase short-chain family member 2; BET: Bromodomain and extra-terminal; CoA: Coenzyme A; LDHA: Lactate dehydrogenase A; MCT: Monocarboxylate transporter; PD-1: Programmed death 1.

Citation: Hawanga GD, Li ZX, Li MX, Zhang XL, Qian DH. Lactylation in pancreatic cancer: From molecular mechanisms to therapeutic perspectives. World J Clin Oncol 2026; 17(3): 115882
URL: https://www.wjgnet.com/2218-4333/full/v17/i3/115882.htm
DOI: https://dx.doi.org/10.5306/wjco.v17.i3.115882

Hawanga GD, Li ZX, Li MX, Zhang XL, Qian DH. Lactylation in pancreatic cancer: From molecular mechanisms to therapeutic perspectives. World J Clin Oncol 2026; 17(3): 115882 [DOI: 10.5306/wjco.v17.i3.115882]

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