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©The Author(s) 2025.
World J Clin Oncol. May 24, 2025; 16(5): 105322
Published online May 24, 2025. doi: 10.5306/wjco.v16.i5.105322
Published online May 24, 2025. doi: 10.5306/wjco.v16.i5.105322
Table 5 Comparison of relative invasive capacities in each group
Group | mean ± SD | vs group | 95%CI | F | η2 |
Control | 39.20 ± 4.00b,c,d | H. pylori | -49.44 to -34.16 | 514.75 | 0.990 |
si-DMBT1 | -90.04 to -74.76 | ||||
H. pylori + si-DMBT1 | -143.04 to -127.76 | ||||
H. pylori | 81.00 ± 3.54a,c,d | si-DMBT1 | -48.24 to -32.96 | ||
H. pylori + si-DMBT1 | -101.24 to -85.96 | ||||
si-DMBT1 | 121.60 ± 5.23a,b,d | H. pylori + si-DMBT1 | -60.64 to -45.36 | ||
H. pylori + si-DMBT1 | 174.60 ± 8.62a,b,c |
- Citation: Zhou X, Wang LQ, Song S, Xu M, Li CP. Helicobacter pylori infection promotes the progression of gastric cancer by regulating the expression of DMBT1. World J Clin Oncol 2025; 16(5): 105322
- URL: https://www.wjgnet.com/2218-4333/full/v16/i5/105322.htm
- DOI: https://dx.doi.org/10.5306/wjco.v16.i5.105322