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©The Author(s) 2025.
World J Clin Oncol. May 24, 2025; 16(5): 105322
Published online May 24, 2025. doi: 10.5306/wjco.v16.i5.105322
Published online May 24, 2025. doi: 10.5306/wjco.v16.i5.105322
Table 3 Comparison of relative proliferative capacities in each group
Group | mean ± SD | vs group | 95%CI | F | η2 |
Control | 0.68 ± 0.05b,c,d | H. pylori | -0.20 to -0.09 | 47.84 | 0.878 |
si-DMBT1 | -0.25 to -0.14 | ||||
H. pylori + si-DMBT1 | -0.38 to -0.27 | ||||
H. pylori | 0.82 ± 0.06a,d | si-DMBT1 | -0.11 to 0.01 | ||
H. pylori + si-DMBT1 | -0.24 to -0.12 | ||||
si-DMBT1 | 0.87 ± 0.04a,d | H. pylori + si-DMBT1 | -0.19 to -0.07 | ||
H. pylori + si-DMBT1 | 1.00 ± 0.03a,b,c |
- Citation: Zhou X, Wang LQ, Song S, Xu M, Li CP. Helicobacter pylori infection promotes the progression of gastric cancer by regulating the expression of DMBT1. World J Clin Oncol 2025; 16(5): 105322
- URL: https://www.wjgnet.com/2218-4333/full/v16/i5/105322.htm
- DOI: https://dx.doi.org/10.5306/wjco.v16.i5.105322