Gastric organoids: A promising model for studying “inflammation-cancer” transition in atrophic gastritis

doi:10.5306/wjco.v16.i11.110453

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World Journal of Clinical Oncology

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2218-4333

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Clin Oncol. Nov 24, 2025; 16(11): 110453
Published online Nov 24, 2025. doi: 10.5306/wjco.v16.i11.110453

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Figure 2 Role of Helicobacter pylori in inflammation-cancer transition. P13K: Phosphoinositide 3-kinase; AKT: Protein kinase B; CagA: Cytotoxin-associated protein A; MAPK: Mitogen-activated protein kinase; VacA: Vacuolating cytotoxin A; ROS: Reactive oxygen species; Nrf2: Nuclear factor erythroid-2-related factor 2; FOXO: Forkhead box O; NF-κB: Nuclear factor kappa-light-chain-enhancer of activated B cells; STAT3: Signal transducer and activator of transcription 3; IL: Interleukin; EMT: Epithelial-mesenchymal transition.

Citation: Liu C, Wu CH, Jia YB, Qiu JX, Li XY, Ling JH. Gastric organoids: A promising model for studying “inflammation-cancer” transition in atrophic gastritis. World J Clin Oncol 2025; 16(11): 110453
URL: https://www.wjgnet.com/2218-4333/full/v16/i11/110453.htm
DOI: https://dx.doi.org/10.5306/wjco.v16.i11.110453

Liu C, Wu CH, Jia YB, Qiu JX, Li XY, Ling JH. Gastric organoids: A promising model for studying “inflammation-cancer” transition in atrophic gastritis. World J Clin Oncol 2025; 16(11): 110453 [PMID: 41355914 DOI: 10.5306/wjco.v16.i11.110453]

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