Molecular targets and mechanisms of different aberrant alternative splicing in metastatic liver cancer

doi:10.5306/wjco.v15.i4.531

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World Journal of Clinical Oncology

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Clin Oncol. Apr 24, 2024; 15(4): 531-539
Published online Apr 24, 2024. doi: 10.5306/wjco.v15.i4.531

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Figure 1 Seven types of variable splicing in vivo. (1) ES: Exon skip; (2) RI: Retained intron; (3) AD: Alternate Donor site; (4) AA: Alternate acceptor site; (5) AP: Alternate promoter; (6) AT: Alternate terminator; (7) ME: Mutually exclusive exons.

Citation: Geng DY, Chen QS, Chen WX, Zhou LS, Han XS, Xie QH, Guo GH, Chen XF, Chen JS, Zhong XP. Molecular targets and mechanisms of different aberrant alternative splicing in metastatic liver cancer. World J Clin Oncol 2024; 15(4): 531-539
URL: https://www.wjgnet.com/2218-4333/full/v15/i4/531.htm
DOI: https://dx.doi.org/10.5306/wjco.v15.i4.531