Role of CCM3 in lead-induced neurological damage in diabetes

doi:10.4239/wjd.113938

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World Journal of Diabetes

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1948-9358

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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Copyright: ©Author(s) 2026. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution-NonCommercial (CC BY-NC 4.0) license. No commercial re-use. See permissions. Published by Baishideng Publishing Group Inc.

World J Diabetes. Aug 15, 2026; 17(8): 113938
Published online Aug 15, 2026. doi: 10.4239/wjd.113938

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Figure 4 Fluorescence co-localization results of CD31 and glial fibrillary acidic protein in the hippocampus of mice in each group. A: Expression of CD31 and glial fibrillary acidic protein (GFAP) in the hippocampus of mice in each group, B: Average fluorescence intensity of CD31, C: Average fluorescence intensity of GFAP. ^aP < 0.05 vs wild-type control group, ^cP < 0.05 vs wild-type diabetes group. WT: Wild-type.

Citation: Liang X, Yang LX, Tao T, Ding WJ, Tang XY, Shang ZB, Zhou H, He Y, Sun Y. Role of CCM3 in lead-induced neurological damage in diabetes. World J Diabetes 2026; 17(8): 113938
URL: https://www.wjgnet.com/1948-9358/full/v17/i8/113938.htm
DOI: https://dx.doi.org/10.4239/wjd.113938

Liang X, Yang LX, Tao T, Ding WJ, Tang XY, Shang ZB, Zhou H, He Y, Sun Y. Role of CCM3 in lead-induced neurological damage in diabetes. World J Diabetes 2026; 17(8): 113938 [DOI: 10.4239/wjd.113938]

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