Troxerutin improves diabetic cognitive dysfunction by inhibiting mitochondrial fission mediated by transient receptor potential melastatin 7/calcineurin/dynamin-related protein 1ser637

doi:10.4239/wjd.v16.i8.106833

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Aug 15, 2025 (publication date) through Aug 17, 2025

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World Journal of Diabetes

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1948-9358

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Diabetes. Aug 15, 2025; 16(8): 106833
Published online Aug 15, 2025. doi: 10.4239/wjd.v16.i8.106833

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Figure 1 A schematic diagram illustrating the mechanism of troxerutin in improving diabetic cognitive dysfunction through inhibiting transient receptor potential melastatin 7-mediated mitochondrial fission. HG: High-concentration glucose; ATP: Adenosine triphosphate; TRPM7: Transient receptor potential melastatin 7; CaN: Calcineurin.

Citation: Li J, Gao M, Wang JX, Li HY, Wang P, Yuan F, Liu AJ, Zhang SY. Troxerutin improves diabetic cognitive dysfunction by inhibiting mitochondrial fission mediated by transient receptor potential melastatin 7/calcineurin/dynamin-related protein 1^ser637. World J Diabetes 2025; 16(8): 106833
URL: https://www.wjgnet.com/1948-9358/full/v16/i8/106833.htm
DOI: https://dx.doi.org/10.4239/wjd.v16.i8.106833