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Personal Data Surname Name Organization and Position Date of birth Saulle Irma University of Milan - PhD Department of Biomedical and Clinical Sciences 07/04/1986 After graduation in Biotecnology in 2010 at University of Brescia, I obtained a Fellowship from University of Milan Doctorate in Molecular Medicine, to work at the Department of Immunology, University of Milan from 2011 to 2013 in the field of immune responses in HIV-infected patients. In 2014 I became Assistant Professor in Applied Biology  and Immunology at University of Milan. Research and Professional experience I am a molecular-immunologist. I am an Assistant Professor Professor in Applied Biology and Immunology at the University of Milano, with nearly 10 year experience in academic research and 5 years teaching in Medical and Nursing School. I am skilled in basic laboratory research and in molecular immunology. My research focus on the molecular mechanisms of several chronic pathologies such as autoimmune viral infections mainly HIV-1 infection, and diseases,. I manage a research group working on the molecular mechanisms of natural resistance to HIV-1 infection.   Congress Oral Communications   2016        Italian Conference on AIDS and Retroviruses (ICAR) Oral Presentation           ▪              Milan (Italy) 2016        Conference on Retroviruses and Opportunistic Infections (CROI) Oral presentation      Boston (Massachusetts)                                2015        Italian Conference on AIDS and Retroviruses (ICAR) Oral Presentation           ▪              Riccione(Italy) 2014        Research for Prevention  (R4P) Oral Presentation                 Cape Town (South Africa) 2013   International AIDS society (IAS) Oral presentation;               Kuala Lumpur, (Malesia) 2013        Intenational Congress of Immunology (Oral presentation)               ▪              Milan (Italy)         Scientific Publications Total number of publications:  17 h-index: 6   THE INTERLEUKIN 21 (IL 21)/ microRNA-29 (miR-29) AXIS IS ASSOCIATED WITH NATURAL RESISTANCE TO HIV-1 INFECTION. AIDS 2018 accepted for pubblication   High Expression of Antiviral and Vitamin D Pathway Genes Are a Natural Characteristic of a Small Cohort of HIV-1-Exposed Seronegative Individuals. Front Immunol, 2017. ,   Stimulation of PBMC and Monocyte-Derived Macrophages via Toll-Like Receptor Activates Innate Immune Pathways in HIV-Infected Patients on Virally Suppressive Combination Antiretroviral Therapy, Front Immunol, 2016   A 6-amino acid insertion/deletion polymorphism in the mucin domain of TIM-1 confers protections against HIV-1 infection, Microbs Infections, 2017   Precursor Forms of Vitamin D Reduce HIV-1 Infection In Vitro, J Acquir Immune Defic Syndr, 2016   Stimulation of PBMC and Monocyte-Derived Macrophages via Toll-Like Receptor Activates Innate Immune Pathways in HIV-Infected Patients on Virally Suppressive Combination Antiretroviral Therapy, Front Immunol, 2016   Thiazolides Elicit Anti-Viral Innate Immunity and Reduce HIV Replication, Sci Rep, 2016   Identification of a Specific miRNA Profile in HIV-Exposed Seronegative Individuals, J Acquir Immune Defic Syndr, 2016   Upregulation of inflammasome activity and increased gut permeability are associated with obesity in children and adolescents, Int J Obes (Lond), 2016   Immune Activation Is Present in HIV-1-Exposed Seronegative Individuals and Is Independent of Microbial Translocation.AIDS Res Hum Retroviruses 2016   Variants in the CYP7B1 gene region do not affect natural resistance to HIV-1 infection, Retrovirology, 2015     ARegulatory Polymorphism in HAVCR modulates Susceptibility to HIV-1 infection, Plos One, 2014   Vitamin D receptor gene polymorphisms are associated with obesity and inflammosome activity, Plos One, 2014   Evolutionary Analysis Identifies an MX2 Haplotype Associated with Natural Resistance to HIV-1 Infection, Molecular Biology and Evolution, 2014   Endoplasmic reticulum aminopeptidase 2 haplotypes play a role in modulating susceptibility to HIV infection, AIDS, 2013     Genetic variability at the TREX1 locus is not associated with natural resistance to HIV-1 infection , AIDS, 2012   A common polymorphism in TLR3 confers natural resistance to HIV-1 infection. J Immunol. 2012