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Cited by in F6Publishing
For: Squitti R, Faller P, Hureau C, Granzotto A, White AR, Kepp KP. Copper Imbalance in Alzheimer's Disease and Its Link with the Amyloid Hypothesis: Towards a Combined Clinical, Chemical, and Genetic Etiology. J Alzheimers Dis 2021;83:23-41. [PMID: 34219710 DOI: 10.3233/JAD-201556] [Cited by in Crossref: 1] [Cited by in F6Publishing: 1] [Article Influence: 1.0] [Reference Citation Analysis]
Number Citing Articles
1 Metsla K, Kirss S, Laks K, Sildnik G, Palgi M, Palumaa T, Tõugu V, Palumaa P. α-Lipoic Acid Has the Potential to Normalize Copper Metabolism, Which Is Dysregulated in Alzheimer's Disease. J Alzheimers Dis 2021. [PMID: 34864665 DOI: 10.3233/JAD-215026] [Reference Citation Analysis]
2 Kim N, Lee HJ. Redox-Active Metal Ions and Amyloid-Degrading Enzymes in Alzheimer's Disease. Int J Mol Sci 2021;22:7697. [PMID: 34299316 DOI: 10.3390/ijms22147697] [Reference Citation Analysis]
3 Pal A, Rani I, Pawar A, Picozza M, Rongioletti M, Squitti R. Microglia and Astrocytes in Alzheimer's Disease in the Context of the Aberrant Copper Homeostasis Hypothesis. Biomolecules 2021;11:1598. [PMID: 34827595 DOI: 10.3390/biom11111598] [Reference Citation Analysis]
4 Metaxas A. Imbalances in Copper or Zinc Concentrations Trigger Further Trace Metal Dyshomeostasis in Amyloid-Beta Producing Caenorhabditis elegans. Front Neurosci 2021;15:755475. [PMID: 34707479 DOI: 10.3389/fnins.2021.755475] [Reference Citation Analysis]