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Cited by in F6Publishing
For: Mou J, Liu X, Pei D. Overexpression of C-terminal fragment of glutamate receptor 6 prevents neuronal injury in kainate-induced seizure via disassembly of GluR6-PSD-95-MLK3 signaling module. Neural Regen Res 2014;9:2059-65. [PMID: 25657722 DOI: 10.4103/1673-5374.147932] [Cited by in Crossref: 4] [Cited by in F6Publishing: 3] [Article Influence: 0.6] [Reference Citation Analysis]
Number Citing Articles
1 Craige SM, Reif MM, Kant S. Mixed – Lineage Protein kinases (MLKs) in inflammation, metabolism, and other disease states. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease 2016;1862:1581-6. [DOI: 10.1016/j.bbadis.2016.05.022] [Cited by in Crossref: 22] [Cited by in F6Publishing: 20] [Article Influence: 3.7] [Reference Citation Analysis]
2 Keller B, García-sevilla JA. Inhibitory effects of imidazoline receptor ligands on basal and kainic acid-induced neurotoxic signalling in mice. J Psychopharmacol 2016;30:875-86. [DOI: 10.1177/0269881116652579] [Cited by in Crossref: 7] [Cited by in F6Publishing: 7] [Article Influence: 1.2] [Reference Citation Analysis]
3 Zhang W, Wang X, Yu M, Li JA, Meng H. The c-Jun N-terminal kinase signaling pathway in epilepsy: activation, regulation, and therapeutics. J Recept Signal Transduct Res 2018;38:492-8. [PMID: 31038026 DOI: 10.1080/10799893.2019.1590410] [Cited by in Crossref: 2] [Cited by in F6Publishing: 2] [Article Influence: 0.7] [Reference Citation Analysis]