Overproduction of nitric oxide inhibits vascular reactivity in portal hypertensive rats

doi:10.3748/wjg.v3.i4.221

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Dec 15, 1997 (publication date) through Apr 23, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Dec 15, 1997; 3(4): 221-224
Published online Dec 15, 1997. doi: 10.3748/wjg.v3.i4.221

Table 4 Rmax and EC₅₀ values for KCl and phenylephrine in aortic rings from rats with portal hypertension and sham-operated control rats with and without L-NNA treatment (x ± s_x-)

Agent	EC₅₀		Maximal contractions (g)
Agent	Untreated	L-NNA	Untreated	L-NNA
KCl (mmol/L)
PH	26.5 ± 0.9^b^a	20.2 ± 0.8	0.72 ± 0.05^b^a	1.08 ± 0.1^b
Control	22.3 ± 1.7^a	19.4 ± 1.2	0.98 ± 0.05^a	1.21 ± 0.11
PE (× 10^-8 mol/L)
PH	3.72 ± 0.4^b^a	2.24 ± 0.18	1.04 ± 0.08^b^a	1.43 ± 0.14^a
Control	2.81 ± 0.2^b	2.18 ± 0.14	1.34 ± 0.39^a	1.72 ± 0.11

^bP < 0.01 vs control;

^aP < 0.05 vs L-NNA.

Citation: Li XR, Wu JS, He ZS, Ma QJ, Gao DM. Overproduction of nitric oxide inhibits vascular reactivity in portal hypertensive rats. World J Gastroenterol 1997; 3(4): 221-224
URL: https://www.wjgnet.com/1007-9327/full/v3/i4/221.htm
DOI: https://dx.doi.org/10.3748/wjg.v3.i4.221