Bile-acid-activated farnesoid X receptor regulates hydrogen sulfide production and hepatic microcirculation

doi:10.3748/wjg.15.2097

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May 7, 2009 (publication date) through Apr 23, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. May 7, 2009; 15(17): 2097-2108
Published online May 7, 2009. doi: 10.3748/wjg.15.2097

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Figure 5 FXR loss of function sensitizes mice to CCl₄-induced liver fibrosis. A: Sirius red staining of liver section obtained from FXR +/+ mice; B: Sirius red staining of liver section obtained from FXR -/- mice; C: Sirius red staining of liver section obtained from FXR +/+ mice treated with CCl₄; D: Sirius red staining of liver section obtained from FXR -/- mice treated with CCl₄; E: Liver section stained with CSE monoclonal antibody obtained from FXR +/+ mice; F: Liver section stained with CSE monoclonal antibody obtained from FXR -/- mice; G: Liver section stained with CSE monoclonal antibody obtained from FXR +/+ mice treated with CCl₄; H: Liver section stained with CSE monoclonal antibody obtained from FXR -/- mice treated with CCl₄.

Citation: Renga B, Mencarelli A, Migliorati M, Distrutti E, Fiorucci S. Bile-acid-activated farnesoid X receptor regulates hydrogen sulfide production and hepatic microcirculation. World J Gastroenterol 2009; 15(17): 2097-2108
URL: https://www.wjgnet.com/1007-9327/full/v15/i17/2097.htm
DOI: https://dx.doi.org/10.3748/wjg.15.2097