Clinical Research
Copyright ©2006 Baishideng Publishing Group Co.
World J Gastroenterol. Sep 28, 2006; 12(36): 5853-5858
Published online Sep 28, 2006. doi: 10.3748/wjg.v12.i36.5853
Figure 1
Figure 1 Mean hepatic vein velocity in study populations. It was significantly higher in cirrhotic patients than non-cirrhotic controls. (mean ± SD; bP < 0.0001 vs NLP and CH).
Figure 2
Figure 2 Mean hepatic vein velocity in study population. Hepatic vein velocity in cirrhotic patients according to Child-Pugh group (mean ± SD; bP < 0.0001 vs A and B).
Figure 3
Figure 3 Maximum forward velocity of hepatic vein in study populations. (mean ± SD; bP < 0.0001 vs NLP and CH).
Figure 4
Figure 4 Relation between mean hepatic vein velocity and liver function test in cirrhotic patients. A: serum albumin; B: serum bilirubin; C: prothrombin time.
Figure 5
Figure 5 Mean hepatic vein velocity in patients depending upon the accumulation of ascites. (mean ± SD; bP < 0.01 vs nil and moderate to massive).
Figure 6
Figure 6 Correlation between mean hepatic vein velocity and right portal vein flow in cirrhotic patients (A) and controls (B).
Figure 7
Figure 7 Right portal flow in cirrhotic patients. “Very high” group, which we defined as those having velocity ≥ 20 cm/s had increased perfusion of right lobe of the liver.