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For: Vrzalikova K, Sunmonu T, Reynolds G, Murray P. Contribution of Epstein⁻Barr Virus Latent Proteins to the Pathogenesis of Classical Hodgkin Lymphoma. Pathogens 2018;7:E59. [PMID: 29954084 DOI: 10.3390/pathogens7030059] [Cited by in Crossref: 10] [Cited by in F6Publishing: 8] [Article Influence: 2.5] [Reference Citation Analysis]
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3 Wojtak K, Perales-Puchalt A, Weiner DB. Novel Synthetic DNA Immunogens Targeting Latent Expressed Antigens of Epstein-Barr Virus Elicit Potent Cellular Responses and Inhibit Tumor Growth. Vaccines (Basel) 2019;7:E44. [PMID: 31137606 DOI: 10.3390/vaccines7020044] [Cited by in Crossref: 4] [Cited by in F6Publishing: 5] [Article Influence: 1.3] [Reference Citation Analysis]
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6 Schneider J, Volkmer I, Engel K, Emmer A, Staege MS. Expression of A New Endogenous Retrovirus-Associated Transcript in Hodgkin Lymphoma Cells. Int J Mol Sci 2019;20:E5320. [PMID: 31731509 DOI: 10.3390/ijms20215320] [Reference Citation Analysis]
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9 Umakanthan S, Bukelo MM. Molecular Genetics in Epstein-Barr Virus-Associated Malignancies. Life (Basel) 2021;11:593. [PMID: 34206255 DOI: 10.3390/life11070593] [Reference Citation Analysis]
10 Lorenzetti MA, Mosna MJ, De Matteo EN, García Lombardi M, Colli SL, Preciado MV. Overexpression of survivin in pediatric Hodgkin lymphoma tumor cells: Characterization of protein expression and splice-variants transcription profile. Exp Mol Pathol 2019;108:24-31. [PMID: 30876863 DOI: 10.1016/j.yexmp.2019.03.005] [Cited by in Crossref: 1] [Cited by in F6Publishing: 1] [Article Influence: 0.3] [Reference Citation Analysis]
11 Hassani A, Reguraman N, Shehab S, Khan G. Primary Peripheral Epstein-Barr Virus Infection Can Lead to CNS Infection and Neuroinflammation in a Rabbit Model: Implications for Multiple Sclerosis Pathogenesis. Front Immunol 2021;12:764937. [PMID: 34899715 DOI: 10.3389/fimmu.2021.764937] [Reference Citation Analysis]