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Cited by in F6Publishing
For: Huber NL, Bailey SR, Schuster R, Ogle CK, Lentsch AB, Pritts TA. Prior thermal injury accelerates endotoxin-induced inflammatory cytokine production and intestinal nuclear factor-κB activation in mice. J Burn Care Res 2012;33:279-85. [PMID: 22079902 DOI: 10.1097/BCR.0b013e3182331d75] [Cited by in Crossref: 5] [Cited by in F6Publishing: 4] [Article Influence: 0.5] [Reference Citation Analysis]
Number Citing Articles
1 Yun HM, Oh JH, Shim JH, Ban JO, Park KR, Kim JH, Lee DH, Kang JW, Park YH, Yu D, Kim Y, Han SB, Yoon DY, Hong JT. Antitumor activity of IL-32β through the activation of lymphocytes, and the inactivation of NF-κB and STAT3 signals. Cell Death Dis 2013;4:e640. [PMID: 23703385 DOI: 10.1038/cddis.2013.166] [Cited by in Crossref: 38] [Cited by in F6Publishing: 37] [Article Influence: 4.2] [Reference Citation Analysis]
2 Akhzari S, Rezvan H, Zolhavarieh SM. Expression of pro-inflammatory genes in lesions, spleens and blood neutrophils after burn injuries in mice treated with silver sulfodiazine. Iran J Basic Med Sci 2017;20:769-75. [PMID: 28852441 DOI: 10.22038/IJBMS.2017.9008] [Cited by in F6Publishing: 2] [Reference Citation Analysis]
3 Kartchner LB, Gode CJ, Dunn JLM, Glenn LI, Duncan DN, Wolfgang MC, Cairns BA, Maile R. One-hit wonder: Late after burn injury, granulocytes can clear one bacterial infection but cannot control a subsequent infection. Burns 2019;45:627-40. [PMID: 30833100 DOI: 10.1016/j.burns.2018.08.019] [Cited by in Crossref: 5] [Cited by in F6Publishing: 4] [Article Influence: 1.7] [Reference Citation Analysis]
4 Abdullahi A, Amini-Nik S, Jeschke MG. Animal models in burn research. Cell Mol Life Sci 2014;71:3241-55. [PMID: 24714880 DOI: 10.1007/s00018-014-1612-5] [Cited by in Crossref: 188] [Cited by in F6Publishing: 162] [Article Influence: 23.5] [Reference Citation Analysis]