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For:	Luo Q, Song Y, Kang J, Wu Y, Wu F, Li Y, Dong Q, Wang J, Song C, Guo H. mtROS-mediated Akt/AMPK/mTOR pathway was involved in Copper-induced autophagy and it attenuates Copper-induced apoptosis in RAW264.7 mouse monocytes. Redox Biol 2021;41:101912. [PMID: 33706171 DOI: 10.1016/j.redox.2021.101912] [Cited by in Crossref: 2] [Cited by in F6Publishing: 1] [Article Influence: 2.0] [Reference Citation Analysis]

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Luo Q, Song Y, Kang J, Wu Y, Wu F, Li Y, Dong Q, Wang J, Song C, Guo H. mtROS-mediated Akt/AMPK/mTOR pathway was involved in Copper-induced autophagy and it attenuates Copper-induced apoptosis in RAW264.7 mouse monocytes. Redox Biol 2021;41:101912. [PMID: 33706171 DOI: 10.1016/j.redox.2021.101912] [Cited by in Crossref: 2] [Cited by in F6Publishing: 1] [Article Influence: 2.0] [Reference Citation Analysis]

Number	Citing Articles
1	Cui W, Yang X, Chen X, Xiao D, Zhu J, Zhang M, Qin X, Ma X, Lin Y. Treating LRRK2‐Related Parkinson's Disease by Inhibiting the mTOR Signaling Pathway to Restore Autophagy. Adv Funct Mater 2021;31:2105152. [DOI: 10.1002/adfm.202105152] [Cited by in Crossref: 10] [Cited by in F6Publishing: 9] [Article Influence: 10.0] [Reference Citation Analysis]
2	Wu M, Cong Y, Wang K, Yu H, Zhang X, Ma M, Duan Z, Pei X. Bisphenol A impairs macrophages through inhibiting autophagy via AMPK/mTOR signaling pathway and inducing apoptosis. Ecotoxicol Environ Saf 2022;234:113395. [PMID: 35298966 DOI: 10.1016/j.ecoenv.2022.113395] [Reference Citation Analysis]
3	Liao J, Li Q, Hu Z, Yu W, Zhang K, Ma F, Han Q, Zhang H, Guo J, Hu L, Pan J, Li Y, Tang Z. Mitochondrial miR-1285 regulates copper-induced mitochondrial dysfunction and mitophagy by impairing IDH2 in pig jejunal epithelial cells. J Hazard Mater 2022;422:126899. [PMID: 34418838 DOI: 10.1016/j.jhazmat.2021.126899] [Reference Citation Analysis]
4	Xiong R, Li N, Chen L, Wang W, Wang B, Jiang W, Geng Q. STING protects against cardiac dysfunction and remodelling by blocking autophagy. Cell Commun Signal 2021;19:109. [PMID: 34749750 DOI: 10.1186/s12964-021-00793-0] [Reference Citation Analysis]