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For: Suzuki-Hatano S, Saha M, Soustek MS, Kang PB, Byrne BJ, Cade WT, Pacak CA. AAV9-TAZ Gene Replacement Ameliorates Cardiac TMT Proteomic Profiles in a Mouse Model of Barth Syndrome. Mol Ther Methods Clin Dev 2019;13:167-79. [PMID: 30788385 DOI: 10.1016/j.omtm.2019.01.007] [Cited by in Crossref: 7] [Cited by in F6Publishing: 10] [Article Influence: 2.3] [Reference Citation Analysis]
Number Citing Articles
1 Pang J, Bao Y, Mitchell-silbaugh K, Veevers J, Fang X. Barth Syndrome Cardiomyopathy: An Update. Genes 2022;13:656. [DOI: 10.3390/genes13040656] [Reference Citation Analysis]
2 Hanaford AR, Cho YJ, Nakai H. AAV-vector based gene therapy for mitochondrial disease: progress and future perspectives. Orphanet J Rare Dis 2022;17:217. [PMID: 35668433 DOI: 10.1186/s13023-022-02324-7] [Reference Citation Analysis]
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4 Cade WT, Laforest R, Bohnert KL, Reeds DN, Bittel AJ, de Las Fuentes L, Bashir A, Woodard PK, Pacak CA, Byrne BJ, Gropler RJ, Peterson LR. Myocardial glucose and fatty acid metabolism is altered and associated with lower cardiac function in young adults with Barth syndrome. J Nucl Cardiol 2021;28:1649-59. [PMID: 31705425 DOI: 10.1007/s12350-019-01933-3] [Cited by in Crossref: 8] [Cited by in F6Publishing: 7] [Article Influence: 2.7] [Reference Citation Analysis]
5 Cai L, Tong J, Zhang Z, Zhang Y, Jiang L, Hou X, Zhang H. Staphylococcus aureus-induced proteomic changes in the mammary tissue of rats: A TMT-based study. PLoS One 2020;15:e0231168. [PMID: 32365127 DOI: 10.1371/journal.pone.0231168] [Cited by in Crossref: 2] [Cited by in F6Publishing: 1] [Article Influence: 1.0] [Reference Citation Analysis]
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7 Yang Z, Yan C, Liu W, Xu W, Li C, Yan M, Liu B, Zhu Z. Identification of novel autoantibodies in ascites of relapsed paclitaxel-resistant gastric cancer with peritoneal metastasis using immunome protein microarrays and proteomics. Cancer Biomark 2021;31:329-38. [PMID: 33896831 DOI: 10.3233/CBM-203142] [Reference Citation Analysis]
8 Garlid AO, Schaffer CT, Kim J, Bhatt H, Guevara-Gonzalez V, Ping P. TAZ encodes tafazzin, a transacylase essential for cardiolipin formation and central to the etiology of Barth syndrome. Gene 2020;726:144148. [PMID: 31647997 DOI: 10.1016/j.gene.2019.144148] [Cited by in Crossref: 3] [Cited by in F6Publishing: 4] [Article Influence: 1.0] [Reference Citation Analysis]
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10 Suzuki-Hatano S, Sriramvenugopal M, Ramanathan M, Soustek M, Byrne BJ, Cade WT, Kang PB, Pacak CA. Increased mtDNA Abundance and Improved Function in Human Barth Syndrome Patient Fibroblasts Following AAV-TAZ Gene Delivery. Int J Mol Sci 2019;20:E3416. [PMID: 31336787 DOI: 10.3390/ijms20143416] [Cited by in Crossref: 5] [Cited by in F6Publishing: 4] [Article Influence: 1.7] [Reference Citation Analysis]
11 Yang Z, Yan C, Yu Z, He C, Li J, Li C, Yan M, Liu B, Wu Y, Zhu Z. Downregulation of CDH11 Promotes Metastasis and Resistance to Paclitaxel in Gastric Cancer Cells. J Cancer 2021;12:65-75. [PMID: 33391403 DOI: 10.7150/jca.48193] [Reference Citation Analysis]
12 Skopenkova VV, Egorova TV, Bardina MV. Muscle-Specific Promoters for Gene Therapy. Acta Naturae 2021;13:47-58. [PMID: 33959386 DOI: 10.32607/actanaturae.11063] [Cited by in Crossref: 2] [Cited by in F6Publishing: 1] [Article Influence: 2.0] [Reference Citation Analysis]
13 Suzuki-Hatano S, Tsai AC, Daugherty A, Pacak CA. TMT Sample Preparation for Proteomics Facility Submission and Subsequent Data Analysis. J Vis Exp 2020. [PMID: 32568242 DOI: 10.3791/60970] [Cited by in Crossref: 1] [Cited by in F6Publishing: 1] [Article Influence: 0.5] [Reference Citation Analysis]