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For: Wang B, Wu Y, Ge Z, Zhang X, Yan Y, Xie Y. NLRC5 deficiency ameliorates cardiac fibrosis in diabetic cardiomyopathy by regulating EndMT through Smad2/3 signaling pathway. Biochem Biophys Res Commun 2020;528:545-53. [PMID: 32505342 DOI: 10.1016/j.bbrc.2020.05.151] [Cited by in Crossref: 6] [Cited by in F6Publishing: 6] [Article Influence: 3.0] [Reference Citation Analysis]
Number Citing Articles
1 Fang T, Sun S, Zhao B, Dong J, Cao K, Wang W. NLRC5 modulates phenotypic transition and inflammation of human venous smooth muscle cells by activating Wnt/β-catenin pathway via TLR4 in varicose veins. Microvasc Res 2022;143:104405. [PMID: 35835172 DOI: 10.1016/j.mvr.2022.104405] [Reference Citation Analysis]
2 Liu X, Lu F, Chen X. Examination of the role of necroptotic damage-associated molecular patterns in tissue fibrosis. Front Immunol 2022;13:886374. [DOI: 10.3389/fimmu.2022.886374] [Reference Citation Analysis]
3 Ke X, Chen X, Yan L, Zhang Y. Vaspin contributes to autophagy and endothelial-to-mesenchymal transition via PI3K-/AKT-mTOR pathway. Acta Histochem 2022;124:151881. [PMID: 35489106 DOI: 10.1016/j.acthis.2022.151881] [Reference Citation Analysis]
4 Artlett CM. The Mechanism and Regulation of the NLRP3 Inflammasome during Fibrosis. Biomolecules 2022;12:634. [DOI: 10.3390/biom12050634] [Reference Citation Analysis]
5 Wu Y, Peng W, Fang M, Wu M, Wu M. MSCs-Derived Extracellular Vesicles Carrying miR-212-5p Alleviate Myocardial Infarction-Induced Cardiac Fibrosis via NLRC5/VEGF/TGF-β1/SMAD Axis. J Cardiovasc Transl Res 2021. [PMID: 34508321 DOI: 10.1007/s12265-021-10156-2] [Reference Citation Analysis]
6 Tuleta I, Frangogiannis NG. Fibrosis of the diabetic heart: Clinical significance, molecular mechanisms, and therapeutic opportunities. Adv Drug Deliv Rev 2021;176:113904. [PMID: 34331987 DOI: 10.1016/j.addr.2021.113904] [Cited by in Crossref: 4] [Cited by in F6Publishing: 3] [Article Influence: 4.0] [Reference Citation Analysis]
7 Sofias AM, De Lorenzi F, Peña Q, Azadkhah Shalmani A, Vucur M, Wang JW, Kiessling F, Shi Y, Consolino L, Storm G, Lammers T. Therapeutic and diagnostic targeting of fibrosis in metabolic, proliferative and viral disorders. Adv Drug Deliv Rev 2021;175:113831. [PMID: 34139255 DOI: 10.1016/j.addr.2021.113831] [Cited by in Crossref: 5] [Cited by in F6Publishing: 4] [Article Influence: 5.0] [Reference Citation Analysis]
8 Zhang Y, Dong Y, Xiong Z, Zhu Z, Gao F, Wang T, Man W, Sun D, Lin J, Li T, Li C, Zhao Z, Shen M, Sun D, Fan Y. Sirt6-Mediated Endothelial-to-Mesenchymal Transition Contributes Toward Diabetic Cardiomyopathy via the Notch1 Signaling Pathway. Diabetes Metab Syndr Obes 2020;13:4801-8. [PMID: 33324079 DOI: 10.2147/DMSO.S287287] [Cited by in Crossref: 9] [Cited by in F6Publishing: 5] [Article Influence: 4.5] [Reference Citation Analysis]
9 Xian S, Chen A, Wu X, Lu C, Wu Y, Huang F, Zeng Z. Activation of activin/Smad2 and 3 signaling pathway and the potential involvement of endothelial‑mesenchymal transition in the valvular damage due to rheumatic heart disease. Mol Med Rep 2021;23:10. [PMID: 33179113 DOI: 10.3892/mmr.2020.11648] [Cited by in F6Publishing: 1] [Reference Citation Analysis]
10 Wang B, Ge Z, Wu Y, Zha Y, Zhang X, Yan Y, Xie Y. MFGE8 is down-regulated in cardiac fibrosis and attenuates endothelial-mesenchymal transition through Smad2/3-Snail signalling pathway. J Cell Mol Med 2020;24:12799-812. [PMID: 32945126 DOI: 10.1111/jcmm.15871] [Cited by in Crossref: 1] [Cited by in F6Publishing: 4] [Article Influence: 0.5] [Reference Citation Analysis]