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For: Zou W, Wang Y, Ren X, Wang L, Deng S, Mao X, Li Z, Liao Z. TRPV6 variants confer susceptibility to chronic pancreatitis in the Chinese population. Human Mutation 2020;41:1351-7. [DOI: 10.1002/humu.24032] [Cited by in Crossref: 7] [Cited by in F6Publishing: 5] [Article Influence: 3.5] [Reference Citation Analysis]
Number Citing Articles
1 Nett V, Erhardt N, Wyatt A, Wissenbach U. Human TRPV6-pathies caused by gene mutations. Biochim Biophys Acta Gen Subj 2021;1865:129873. [PMID: 33610740 DOI: 10.1016/j.bbagen.2021.129873] [Cited by in Crossref: 1] [Cited by in F6Publishing: 1] [Article Influence: 1.0] [Reference Citation Analysis]
2 Neuberger A, Nadezhdin KD, Sobolevsky AI. Structural mechanisms of TRPV6 inhibition by ruthenium red and econazole. Nat Commun 2021;12:6284. [PMID: 34725357 DOI: 10.1038/s41467-021-26608-x] [Reference Citation Analysis]
3 Masson E, Rebours V, Buscail L, Frete F, Pagenault M, Lachaux A, Chevaux JB, Génin E, Cooper DN, Férec C, Chen JM. The reversion variant (p.Arg90Leu) at the evolutionarily adaptive p.Arg90 site in CELA3B predisposes to chronic pancreatitis. Hum Mutat 2021;42:385-91. [PMID: 33565216 DOI: 10.1002/humu.24178] [Reference Citation Analysis]
4 Mao XT, Deng SJ, Kang RL, Wang YC, Li ZS, Zou WB, Liao Z. Homozygosity of short VNTR lengths in the CEL gene may confer susceptibility to idiopathic chronic pancreatitis. Pancreatology 2021;21:1311-6. [PMID: 34507899 DOI: 10.1016/j.pan.2021.09.001] [Reference Citation Analysis]
5 Hamada S, Masson E, Chen JM, Sakaguchi R, Rebours V, Buscail L, Matsumoto R, Tanaka Y, Kikuta K, Kataoka F, Sasaki A, Le Rhun M, Audin H, Lachaux A, Caumont B, Lorenzo D, Billiemaz K, Besnard R, Koch S, Lamireau T, De Koninck X, Génin E, Cooper DN, Mori Y, Masamune A, Férec C; GREPAN (Genetic REsearch on PANcreatitis) Study Group. Functionally deficient TRPV6 variants contribute to hereditary and familial chronic pancreatitis. Hum Mutat 2021. [PMID: 34923708 DOI: 10.1002/humu.24315] [Reference Citation Analysis]
6 Oracz G, Zaród M, Ewers M, Laumen H, Gambin T, Kamiński P, Grabowska I, Drożak A, Kwiatkowski S, Wertheim-Tysarowska K, Kołodziejczyk E, Domaszewicz A, Dorożko B, Kosińska J, Głuszek S, Kozieł D, Płoski R, Rosendahl J, Witt H, Drożak J, Rygiel AM. Loss of function TRPV6 variants are associated with chronic pancreatitis in nonalcoholic early-onset Polish and German patients. Pancreatology 2021:S1424-3903(21)00569-X. [PMID: 34538581 DOI: 10.1016/j.pan.2021.09.005] [Reference Citation Analysis]
7 Wertheim-Tysarowska K, Oracz G, Rygiel AM. Genetic Risk Factors in Early-Onset Nonalcoholic Chronic Pancreatitis: An Update. Genes (Basel) 2021;12:785. [PMID: 34065437 DOI: 10.3390/genes12050785] [Reference Citation Analysis]
8 Takáts A, Berke G, Szentesi A, Farkas G Jr, Izbéki F, Erőss B, Czakó L, Vincze Á, Hegyi P, Sahin-Tóth M, Hegyi E. Common calcium-sensing receptor (CASR) gene variants do not modify risk for chronic pancreatitis in a Hungarian cohort. Pancreatology 2021;21:1305-10. [PMID: 34481716 DOI: 10.1016/j.pan.2021.08.012] [Reference Citation Analysis]
9 Wang YC, Mao XT, Yu D, Mao SH, Li ZS, Zou WB, Liao Z. Alcohol amplifies the association between common variants at PRSS1-PRSS2 locus and chronic pancreatitis in a dose-dependent manner. Gut 2022:gutjnl-2021-326670. [PMID: 34996825 DOI: 10.1136/gutjnl-2021-326670] [Reference Citation Analysis]