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Cited by in F6Publishing
For: Wang X, Wei Z, Cheng B, Li J, He Y, Lan T, Kemper T, Lin Y, Jiang B, Jiang Y, Meng Z, Lu M. Endoplasmic reticulum stress promotes HBV production by enhancing use of the autophagosome/multivesicular body axis. Hepatology 2022;75:438-54. [PMID: 34580902 DOI: 10.1002/hep.32178] [Cited by in Crossref: 3] [Cited by in F6Publishing: 4] [Article Influence: 3.0] [Reference Citation Analysis]
Number Citing Articles
1 Ye J, Liu X. Interactions between endoplasmic reticulum stress and extracellular vesicles in multiple diseases. Front Immunol 2022;13:955419. [DOI: 10.3389/fimmu.2022.955419] [Reference Citation Analysis]
2 Bhat SA, Kazim SN. HBV cccDNA─A Culprit and Stumbling Block for the Hepatitis B Virus Infection: Its Presence in Hepatocytes Perplexed the Possible Mission for a Functional Cure. ACS Omega. [DOI: 10.1021/acsomega.2c02216] [Reference Citation Analysis]
3 Cao X, Meng P, Shao Y, Yan G, Yao J, Zhou X, Liu C, Zhang L, Shu H, Lu H. Nascent Glycoproteome Reveals That N-Linked Glycosylation Inhibitor-1 Suppresses Expression of Glycosylated Lysosome-Associated Membrane Protein-2. Front Mol Biosci 2022;9:899192. [DOI: 10.3389/fmolb.2022.899192] [Reference Citation Analysis]
4 Wei L, Ploss A. Rise above the stress-Endoplasmic reticulum stress and autophagy enhance the release of hepatitis B virus subparticles. Hepatology 2022;75:248-51. [PMID: 34890054 DOI: 10.1002/hep.32273] [Reference Citation Analysis]